IVNS1ABP
Sign in to saveAlso known as FLARA3, HSPC068, KLHL39, ND1, NS-1, NS1-BP, NS1BP, influenza virus NS1A binding protein
Influenza virus NS1A-binding protein is a protein that in humans is encoded by the IVNS1ABP gene.
In the Vinony graph
Within Vinony's link graph, IVNS1ABP is referenced by 2 other articles, and connects out to PubMed, human chromosome 1 and Ensembl genome database project.
Vinony files it under Genes on human chromosome 1 and Kelch proteins.
Its subject is documented across 6 Wikipedia language editions.
Gene data
IVNS1ABP- Name
- influenza virus NS1A binding protein
- Type
- protein-coding
- Position
- 185,292,931–185,317,862 (−)
- Aliases
- ARA3, FLARA3, HSPC068, IMD70, KLHL39, ND1, NS-1, NS1-BP, NS1BP
- Ensembl
- ENSG00000116679
- RefSeq RNA
- NM_006469.5, NM_016389.2, XM_047434070.1, XM_047434096.1, XM_047434109.1
- RefSeq protein
- NP_006460.2, XP_047290026.1, XP_047290052.1, XP_047290065.1, XP_054189863.1
Involved in RNA splicing; negative regulation of protein ubiquitination; and response to virus. Located in cytosol. Implicated in immunodeficiency 70. [provided by Alliance of Genome Resources, Apr 2022]
Gene Ontology
Biological process
Molecular function
via MyGene.info
Gene · Ensembl
influenza virus NS1A binding protein
- Symbol
- IVNS1ABP
- Biotype
- Protein coding
- Organism
- Homo sapiens
- Location
- 1:185,292,931-185,317,862
- Strand
- Reverse (−)
- Assembly
- GRCh38
via Ensembl · EMBL-EBI
Wikidata facts
- Instance of
- gene
Show 7 more facts
- HomoloGene ID
- 31384
- exact match
- identifiers.org/ncbigene/10625
- found in taxon
- Homo sapiens
- chromosome
- human chromosome 1
- genomic start
- 185265520
- genomic end
- 185317273
- cytogenetic location
- 1q25.3
Sources (4)
via Wikidata · CC0
~1 min read
Encyclopedic overview
2 sectionsContents
- References
- Further reading
Influenza virus NS1A-binding protein is a protein that in humans is encoded by the IVNS1ABP gene.
In melanocytic cells IVNS1ABP gene expression may be regulated by MITF.
Excerpted from Wikipedia’s “IVNS1ABP” article, available under the CC BY-SA 4.0 licence.