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GeneQ18029275· pop 5· linked from 4 articles

Also known as MTDPS6, SYM1, MPV17, mitochondrial inner membrane protein, mitochondrial inner membrane protein MPV17, CMT2EE

Protein MPV17 is a protein that in humans is encoded by the MPV17 gene. It is a mitochondrial inner membrane protein, which has a so far largely unknown role in mtDNA maintenance. Protein MPV17 is expressed in human pancreas, kidney, muscle, liver, lung, placenta, brain and heart. Human MPV17 is the orthologue of the mouse kidney disease gene, Mpv17. Loss of function has been shown to cause hepatocerebral mtDNA depletion syndromes (MDS) with oxidative phosphorylation failure and mtDNA depletion both in affected individuals and in Mpv17−/− mice.

Gene data

MPV17
Name
mitochondrial inner membrane protein MPV17
Type
protein-coding
Aliases
CMT2EE, MTDPS6, SYM1

This gene encodes a mitochondrial inner membrane protein that is implicated in the metabolism of reactive oxygen species. Mutations in this gene have been associated with the hepatocerebral form of mitochondrial DNA depletion syndrome (MDDS). [provided by RefSeq, Jul 2008].

via MyGene.info

Gene · Ensembl

mitochondrial inner membrane protein MPV17

Symbol
MPV17
Biotype
Protein coding
Organism
Homo sapiens
Location
2:27,309,486-27,325,680
Strand
Reverse (−)
Assembly
GRCh38
View on Ensembl →

via Ensembl · EMBL-EBI

Wikidata facts

Show 5 more facts
HomoloGene ID
39746
genomic end
27548547
genomic start
27532360
cytogenetic location
2p23.3
Sources (4)

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~2 min read

Article

9 sections
Contents
  • Function
  • Structure
  • Gene
  • Protein
  • Clinical significance
  • Interactions
  • See also
  • References
  • Further reading

Protein MPV17 is a protein that in humans is encoded by the MPV17 gene. It is a mitochondrial inner membrane protein, which has a so far largely unknown role in mtDNA maintenance. Protein MPV17 is expressed in human pancreas, kidney, muscle, liver, lung, placenta, brain and heart. Human MPV17 is the orthologue of the mouse kidney disease gene, Mpv17. Loss of function has been shown to cause hepatocerebral mtDNA depletion syndromes (MDS) with oxidative phosphorylation failure and mtDNA depletion both in affected individuals and in Mpv17−/− mice.

== Function ==

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