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GeneQ18030697· pop 6· linked from 4 articles

Also known as PP2C-ALPHA, PP2CA, PP2Calpha, protein phosphatase, Mg2+/Mn2+ dependent 1A

Protein phosphatase 1A is an enzyme that in humans is encoded by the PPM1A gene.

Gene data

PPM1A
Name
protein phosphatase, Mg2+/Mn2+ dependent 1A
Type
protein-coding
Aliases
PP2C-ALPHA, PP2CA, PP2Calpha

The protein encoded by this gene is a member of the PP2C family of Ser/Thr protein phosphatases. PP2C family members are known to be negative regulators of cell stress response pathways. This phosphatase dephosphorylates, and negatively regulates the activities of, MAP kinases and MAP kinase kinases. It has been shown to inhibit the activation of p38 and JNK kinase cascades induced by environmental stresses. This phosphatase can also dephosphorylate cyclin-dependent kinases, and thus may be involved in cell cycle control. Overexpression of this phosphatase is reported to activate the expression of the tumor suppressor gene TP53/p53, which leads to G2/M cell cycle arrest and apoptosis. Three alternatively spliced transcript variants encoding distinct isoforms have been described. [provided by RefSeq, Jul 2008].

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Gene · Ensembl

protein phosphatase, Mg2+/Mn2+ dependent 1A

Symbol
PPM1A
Biotype
Protein coding
Organism
Homo sapiens
Location
14:60,245,752-60,299,223
Strand
Forward (+)
Assembly
GRCh38
View on Ensembl →

via Ensembl · EMBL-EBI

Wikidata facts

Image
Protein PPM1A PDB 1a6q.png
Show 5 more facts
HomoloGene ID
56428
genomic end
60765805
genomic start
60712470
cytogenetic location
14q23.1
Sources (4)

via Wikidata · CC0

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Protein phosphatase 1A is an enzyme that in humans is encoded by the PPM1A gene.

The protein encoded by this gene is a member of the PP2C family of Ser/Thr protein phosphatases. PP2C family members are known to be negative regulators of cell stress response pathways. This phosphatase dephosphorylates, and negatively regulates the activities of, MAP kinases and MAP kinase kinases. It has been shown to inhibit the activation of p38 and JNK kinase cascades induced by environmental stresses. This phosphatase can also dephosphorylate cyclin-dependent kinases, and thus may be involved in cell cycle control. Overexpression of this phosphatase is reported to activate the expression of the tumor suppressor gene TP53/p53, which leads to G2/M cell cycle arrest and apoptosis. Three alternatively spliced transcript variants encoding two distinct isoforms have been described.

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